MEDICAL SURGICAL
CARING FOR CLIENTS WITH HEART FAILURE
HEART FAILURE
The heart is a double pump. The right side pumps
deoxygenated blood to the lungs to be oxygenated. The left side pumps that
oxygenated blood into the systemic circulation . If either one of the sides
doesn’t work properly it will effect systemic circulation.
Heart failure is the inability of the heart to
pump a sufficient amount of blood to meet the body’s metabolic needs.
Congestive heart failure (CHF) is the
accumulation of blood and fluid within organs & tissues due to impaired
circulation
HEART FAILURE
Ejection fraction is an estimation of the heart’s efficiency as
a pump. It is a measurement of the % of blood that the left ventricle ejects
when it contracts
Normal is 55% or greater
CLASSIFICATION OF HEART FAILURE – table 34-1
ACUTE - a sudden change in the heart’s ability
to contract that can lead to life threatening symptoms and pulmonary edema
CHRONIC – occurs when some other chronic
condition gradually comprises the heart’s ability to pump effectively
Left and Right Sided Heart Failure
LEFT SIDED HEART FAILURE – when the left
ventricle does not eject blood into the aorta normally
RIGHT SIDED HEART FAILURE – when the right
ventricle fails to eject it’s total filling volume into the pulmonary
artery, causing congestion in the venous vascular system. Most common
cause of right sided heart failure is left sided heart failure
Pathophysiology & Etiology
Inability of the heart muscle to contract forcefully because of
direct damage to the heart wall:
Acute MI
When the pumping chambers enlarge & weaken & are unable to
eject all the blood received
Cardiomyopathy
HTN
Diagnostic Findings
Left sided failure
cxr shows cardiac enlargement
echocardiogram shows enlarged left ventricle,
decreased ejection fraction
MUGA scan measures the ejection fraction more
accurately
Right sided failure: all show enlarged right
ventricle
cxr
echocardiogram
ECG
LEFT SIDED FAILURE
Decreased cardiac output
Blood backs up into the left atrium, & finally the pulmonary
vasculature causing impaired gas exchange
Causes: HTN, tachydysrhythmias, valvular disease,
cardiomyopathy & renal failure
RIGHT-SIDED FAILURE
Blood backs up into the right atrium, superior & inferior vena
cava, & then the venous vasculature
Causes: Most common cause is left-sided heart failure,
MI, respiratory diseases
cor pulmonale – when the heart is affected by lung disease such
as COPD, asthma & can lead to
pulmonary HTN – vasoconstriction in the arterial pulmonary
circulation
Compensatory Mechanisms
These mechanism usually make matters worse by increasing the
amount of blood the heart must pump & the resistance it must overcome from
arterial constriction
Assessment Findings:
Left-sided Failure
Hypoxemia which leads to:
exertional dyspnea
orthopnea
paroxysmal nocturnal dyspnea
hemoptysis
Assessment Findings:
Right-sided Failure
gradual wt gain from fluid retention
dependent pitting edema
ascites
hepatomegaly
jugular vein distention
enlarged right ventricle on chest xray
Medical Management
Focuses on reducing the workload of the heart and improving
cardiac output
Fluid restriction
Low Na+ diet
Drugs-see table 34-1
Digitalization – initially using large doses of digoxin
to build up therapeutic drug levels
diuretics
vasodilators
B-type Natriuretic Peptide - BNP
A cardioprotective neurohormone that functions to decrease BP
by increasing excretion of Na+ & H2O, promoting arterial dilation, &
counteracting renin, angiotension, & aldosterone
BNP is measured via a blood test to estimate how far advanced
the CHF is
See box 34-2
Medical Management
Cardiac Resynchronization Therapy (CRT) – used to restore
synchronization to the right & left ventricles via a pacemaker thereby
improving force of contraction & cardiac ejection fraction
Intra-aortic balloon pump (IABP) – fig 34-4 – a temporary
secondary circulatory pump
Surgical Management
Ventricular Assist Device (LVAD) – fig 34-5 – an auxiliary pump
that supplements the heart’s ability to eject blood. Used for those awaiting
heart transplants
Cardiomyoplasty – a surgical procedure in which the client’s
own chest muscle is grafted to the aorta & wrapped around the heart
augmenting the ineffective heart muscle contraction fig 34-6
Artificial Heart – used for adults < 55 who are waiting heart
transplant – usually temporary – fig 34-9
NURSING MANAGEMENT
Medication administration and teaching
Hx of symptoms
Baseline physical assessment
Daily wts
Accurate I & O
Watch electrolytes & other labs
Measure abdominal girth
See client & family teaching for Heart Failure
34-1
Nursing process – the client with CHF
PULMONARY EDEMA
Fluid accumulation in the lungs that interferes with gas
exchange
An acute emergency
S/S – sudden dyspnea, wheezing, orthopnea, restlessness, cough
often with pink, frothy sputum, cyanosis, tachycardia & severe apprehension,
moist, gurgly respirations
MEDICAL MANAGEMENT
DRUGS
Inatropic agents used to improve myocardial
contractility: dopamine, dobutamine, Inocor, or digitalis
To reduce venous return: diuretics & promote
vasodilation: nitrates, ACE inhibitors, calcium channel blockers
IV morphine to reduce anxiety
OXYGENATION usually by mask or if respiratory
failure occurs may be intubated & put on mechanical ventilator
Nursing Management
IV access
Medication administration
Pulse oximetry
V/S with cardiac monitoring
Foley catheter insertion for accurate monitoring of I&O
GENERAL NUTRITIONAL CONSIDERATIONS
Clients with severe heart failure may require Na+ restriction
to as little as 500-1000mg/day
Clients with mild heart failure may tolerate 3000mg Na+/day
Encourage weight loss in obese clients
Provide 5-6 small meals a day to reduce dyspnea & nausea
related to enlarged abdominal organs
GENERAL PHARMALOGICAL CONSIDERATIONS
Mild heart failure usually responds to thiazide diuretics such
as HCTZ or Hygroton
Severe heart failure usually requires loop diuretics such as
Lasix
For those who don’t respond to diuretics & digitalis ACE
inhibitors are used
GENERAL GERONTOLOGIC CONSIDERATIONS
Dyspnea on exertion is the earliest s/s of heart failure in the
elderly + change in mental status such as confusion
Older adults who take digitalis are at an increased risk for
toxicity due to reduced kidney function & decreased drug excretion
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